20 Apr 2022 11:34 am
New Cause of COVID-19 Blood Clots Identifiedfreeman » 19 Apr 2022, 12:25 pm » wrote: ↑ https://www.thelibertybeacon.com/embalm ... ery-clots/
"Embalmers Find Veins & Arteries Filled With Rubbery Clots"
https://rumble.com/embed/vqqmda/?pub=4
https://rumble.com/embed/vs4dze/?pub=4
"Long, Fibrous Blood Clots Found in Autopsies of the Vaxxed"
https://dailyexpose.uk/2022/01/28/embal ... ery-clots/
A study reveals the virus triggers production of antibodies circulating through the blood, causing clots in people hospitalized with the disease.
https://labblog.uofmhealth.org/lab-repo ... identified
Blood clots continue to wreak havoc for patients with severe COVID-19 infection, and a new study explains what may spark them in up to half of patients.
The culprit: an autoimmune antibody that’s circulating in the blood, attacking the cells and triggering clots in arteries, veins, and microscopic vessels. Blood clots can cause life-threatening events like strokes. And, in COVID-19, microscopic clots may restrict blood flow in the lungs, impairing oxygen exchange.
Outside of novel coronavirus infection, these clot-causing antibodies are typically seen in patients who have the autoimmune disease antiphospholipid syndrome. The connection between autoantibodies and COVID-19 was unexpected, says co-corresponding author Yogen Kanthi, M.D., an assistant professor at the Michigan Medicine Frankel Cardiovascular Center and a Lasker Investigator at the National Institutes of Health’s National Heart, Lung, and Blood Institute.
“In patients with COVID-19, we continue to see a relentless, self-amplifying cycle of inflammation and clotting in the body,” Kanthi says. “Now we’re learning that autoantibodies could be a culprit in this loop of clotting and inflammation that makes people who were already struggling even sicker.
”‘Some of the worst clotting we’ve ever seen’
Co-corresponding author Jason Knight, M.D., Ph.D., a rheumatologist at Michigan Medicine, has been studying antiphospholipid syndrome antibodies in the general population for years.
“Half of the patients hospitalized with COVID-19 were positive for at least one of the autoantibodies, which was quite a surprise,” says Knight, also an associate professor of internal medicine and a leading expert on diseases caused by autoantibodies.
In the new Science Translational Medicine publication, they found about half of the patients who were very sick with COVID-19 were exhibiting a combination of high levels of both the dangerous antibodies and super-activated neutrophils, which are destructive, exploding white blood cells. In April, the team was the first to report that patients hospitalized for severe COVID-19 had higher levels of neutrophil extracellular traps in their blood.